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ACTH 11-24
CAS No. 4237-93-8
ACTH 11-24 ( Adrenocorticotropic Hormone (11-24) )
产品货号. M29947 CAS No. 4237-93-8
ACTH (11-24) is a fragment of adrenocorticotrophin, induces cortisol release. ACTH (11-24) has been identified as a competitive antagonist of ACTH (1-39) and ACTH (1-10) in isolated adrenal cells.
纯度: >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| 规格 | 价格/人民币 | 库存 | 数量 |
| 5MG | ￥806 | 有现货 |
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| 10MG | ￥1102 | 有现货 |
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| 100MG | 获取报价 | 有现货 |
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| 200MG | 获取报价 | 有现货 |
|
| 500MG | 获取报价 | 有现货 |
|
生物学信息
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产品名称ACTH 11-24
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注意事项本公司产品仅用于科研实验，不得用于人体或动物的临床与诊断
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产品简述ACTH (11-24) is a fragment of adrenocorticotrophin, induces cortisol release. ACTH (11-24) has been identified as a competitive antagonist of ACTH (1-39) and ACTH (1-10) in isolated adrenal cells.
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产品描述ACTH (11-24) is a fragment of adrenocorticotrophin, induces cortisol release. ACTH (11-24) has been identified as a competitive antagonist of ACTH (1-39) and ACTH (1-10) in isolated adrenal cells.(In Vitro):ACTH (11-24) acts as an antagonist of adrenocorticotropic hormone receptor. ACTH (11-24) induces cortisol secretion submaximally in freshly dispersed or cultured beef adrenal cortical cells. The maximal cortisol release by ACTH (11-24) is enhanced by forskolin but inhibited by calcium channel blockers.(In Vivo):ACTH (11-24) blocks electroacupuncture (EA) anti-edema but not EA anti-hyperalgesia in rats.
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体外实验ACTH (11-24) elicits cortisol secretion submaximally in freshly dispersed or cultured beef adrenal cortical cells.
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体内实验ACTH (11-24) has slight influences on circulating plasma corticosterone values and on fighting behavior.
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同义词Adrenocorticotropic Hormone (11-24)
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通路Others
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靶点Other Targets
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受体Adrenocorticotropic hormone receptor
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研究领域——
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适应症——
化学信息
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CAS Number4237-93-8
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分子量1652.04
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分子式C77H134N24O16
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纯度>98% (HPLC)
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溶解度In Vitro:?H2O : 100 mg/mL (60.53 mM)
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SMILES——
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化学全称Sequence:Lys-Pro-Val-Gly-Lys-Lys-Arg-Arg-Pro-Val-Lys-Val-Tyr-Pro
运输与储存
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储存条件(-20℃)
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运输条件With Ice Pack
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稳定性≥ 2 years
参考文献
Li ZG, et al. Adrenocorticotropin(1-10) and -(11-24) promote adrenal steroidogenesis by different mechanisms. Endocrinology. 1989 Aug;125(2):592-6.
